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Michael has presented at over one hundred technical and business conferences in more than fifty countries around the world and on all seven continents, including the first ever IT conference in Antarctica. Currently a partner at Convergent Computing (www. San Francisco Bay area, Michael’s writing and extensive public- speaking experience across all seven continents leverage his real- world expertise in helping organizations realize business value from Information Technology infrastructure. Guy Yardeni, MCITP, CISSP, MVP, is an accomplished infrastructure architect, author, and overall geek- for- hire. Guy has been working in the IT industry for more than 1. Guy is an expert at connecting business requirements to technology solutions and driving to successful completion the technical details of the effort while maintaining overall goals and vision. Guy maintains a widely read technical blog at www. Windows MVP. 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A1 - Markers associated with arteriovascular events and methods of use thereof MARKERS ASSOCIATED WITH ARTERIOVASCULAR EVENTSAND METHODS OF USE THEREOFINCORPORATION BY REFERENCEThis application claims. U. S. Provisional Application Ser. No. 6. 0/8. 11,9. June 7, 2. 00. 6. Each of the applications and patents cited in this text, as well as each document or reference cited in each of the applications and patents (including during'the prosecution of each issued paterit; . More generally, documents or references are cited in this text, either in a Reference List before the claims, or in the text itself; and, each of these documents or references (. Documents incorporated by reference into this text may be employed in the practice of the invention. FIELD OF THE INVENTIONThe present invention relates generally to the identification of biological markers associated with arteriovascular events and methods of using such biological markers in screening, prevention, diagnosis, therapy, monitoring, and prognosis of arteriovascular disease. BACKGROUND OF THE INVENTIONArteriovascular disease continues to be a leading cause of morbidity and mortality among adults in Europe and North America. Although age- adjusted death rates have declined over the past two decades, the absolute mortality rate from arteriovascular disease has not. Arteriovascular disease accounts for over one- half million deaths (1 out of every 5) in- the U. S. The lifetime risk of arteriovascular disease after age 4. Even for those who survive to age 7. Arteriovascular diseases incl'ude atherosclerosis and atherothrombosis. Identified risk factors for arteriovascular disease include independent risk factors, such as cigarette smoking, elevated blood pressure (hypertension), elevated serum total cholesterol.(CHOL) and low- density lipoprotein (LDL) cholesterol, low serum high- density lipoprotein (HDL) cholesterol, diabetes mellitus, and advancing age. Conditipnal risk factors for arteriovascular disease include elevated serum triglycerides (TRIG), small LDL particles, elevated serum homocysteine levels, elevated serum lipoprotein (a) (LPA), prothrombotic factors such as fibrinogen (FGA), and inflammatory markers like C- reactive protein (CRP), whose contribution to risk may vary upon their relationship to other identified risk factors. Other risk factors include obesity (measured by weight (WT), height (HT), Body Mass Index (BMI), and abdominal girth comparisons such as waist (. These risk factors have been incorporated into useful predictive models of future arteriovascular events, such as the Framingham Risk Score presented in Wilson, P. W, et al (1. 99. 8) Circulation 9. In particular, while such approaches may, at typical clinical measurement cut- off levels, be relatively sensitive to individuals who have multiple risk factors, experienced past arteriovascular events or who have already confirmed arteriovascular disease (who would be . In the typical adult population, these algorithms yield many more false positives than true positives, particularly in the low (< 6% ten year risk of an acute event) and intermediate risk (6 - 2. While performance metrics for global risk assessment indices may evidence high clinical utility in the population in which the index algorithm was trained, occasionally exhibiting an AUC as high as 0. AUC around 0. 7 (Wilson et al. Framingham Risk Score), such predictive models show relatively low transferability between populations, which may differ based on genetic and other factors, and absent substantial recalibration and re- optimization, often the AUC will drop to below 0. They also are often difficult for clinicians to effectively implement and perform within an active clinical environment, involving complex calculations and numerical manipulations: Thus, the general concept of applying one or more biomarkers to the task of classifying current and predicting future arteriovascular disease or risk of future arteriovascular events is not new in the clinical practice, literature or patent art. Several specific biomarkers, biomarker combinations, and methods have been proposed over time, with limited adoption to date due to several issues including technical difficulty, analytical performance, clinical performance, reliability, and practical clinician application of complex algorithms combining more than one such biomarker. By way of example, Ridker, P. Although certain of the individual panels of biomarkers disclosed therein may be useful in such applications, it is unlikely that the panels, algorithms and analytical processes disclosed therein, selected and trained on past events and known symptomatic disease, will successfully predict the future risk of cardiovascular events in asymptomatic and previously undiagnosed subjects with as high a degree of diagnostic accuracy as is presented and claimed in Tabibiazar over a specific multi- year time horizon, absent substantial and predictive model re- training, re- modeling, optimization and re- purposing likely riot possible absent inputs from such longitudinal studies, which may include changes to cutoffs, reference values and other formula. Although overlap of certain individual biomarkers disclosed in Tabibiazar with individual biomarkers and a subset of the panels of the current invention is acknowledged, each of the individual biomarkers mentioned in Tabibiazar which are also claimed herein in specific, panel combinations of the current invention (andspecifically CCL2, IGF 1, LEP, VEGF, and IL8) were also previously disclosed in the prior published art as associated with cardiovascular disease (each of them were notably mentioned and reviewed in the . Anderson reference, amongst others). Such specific clinical applications, additional biomarkers, specific biomarker combination panels, study designs, and analytical techniques and formula are key aspects of the current invention. Recently, several studies in the scientific literature have been published examining various individual and multiple biomarker strategies, most notably Folsom, A. R. Intern Med 1. 66: 1. Wang, T. J. These studies, utilizing retrospective samples from longitudinal clinical studies such as the Atherosclerosis Risk in Communities Study and the Framingham Heart Study, combined subject clinical parameters and traditional laboratory risk factors (including using such traditional laboratory based biomarkers such as CHOL, CRP, FGA, HDLC, LPA, and Homocysteine), as well as novel markers such as Albumin- to- creatine ratios, Aldosterone, ANP (NPPA), BNP (NPPB), D- dimer, ICAM1, IL6, LEP, MMPI, PLA2. G7, PLAT, PLG, REN, SELE, SERPINEI, TIMPI, THBD, amongst others, both as individual markers and incrementally as additions to multi- marker indices. Both studies found little improvement in the ability to predict future arteriovascular events with novel markers over the models incorporating the basic clinical parameters and traditional laboratory risk factors. As a result, the use of such novel'markers remains clinically controversial. Given the foregoing, it is clear that an important discrepancy has arisen in understanding the role of the aforementioned risk factors and biomarkers compared to the development of arteriovascular disease events. In contrast to the relative ease of recognition and clarity of treatment and prevention strategies in patients with symptomatic arteriovascular disease (i. TIAs) or mild cognitive impairment (MCI), a major problem of detection, treatment, and prevention of arteriovascular disease exists in the large, apparently healthy, population who have no symptoms, yet are at an increased risk to develop arteriovascular disease or experience major arteriovascular events. Alarge number of victims of the disease who are apparently healthy die or have initial acute arteriovascular events suddenly without prior symptoms. Despite the many available risk assessment approaches, a substantial gap remains in the detection of asymptomatic individuals who ultimately develop arteriovascular disease. Currently available screening and diagnostic methods are insufficient to identify asymptomatic individuals before such acute events associated with arteriovascular disease occur. Of those who experience a major arteriovascular event as many as 2. There remains an unmet need in the art to directly diagnose and predict the risk of arteriovascular disease and events, particularly in those individuals who do not exhibit symptoms or few or none of the traditional risk factors currently measured by physicians.' All of the foregoing references, including Tabibiazar, are herein referred to and incorporated in their entirety. SUMMARY OF THE INVENTIONThe present invention relates in part to the discovery that certain biological markers, such as proteins, nucleic acids, polymorphisms, metabolites, and other analytes, as well as certain physiological conditions and states, are present in subjects with an increased risk of arteriovascular events, such as, but not limited to, acute coronary syndromes such as myocardial infarction and unstable angina, as well as other acute events associated with an arteriovascular disease, including those associated with atherosclerosis, atherothrombosis, coronary artery disease (CAD), peripheral artery disease (PAD), and cerebrovascular disease (CVD), but where such subjects do not exhibit some or all of the traditional risk factors of these diseases, or subjects who are asymptomatic for these diseases. Accordingly, the invention provides biological markers of arteriovascular events that, when used together in combinations of three or more such biomarker combinations, or . 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